PART 2: Cardinal Manifestations and Presentation of Diseases
SECTION 1 Pain
14 Headache
 | Figure 14-1 Brainstem pathways that modulate sensory input. The key pathway for pain in migraine is the trigeminovascular input from the meningeal vessels, which passes through the trigeminal ganglion and synapses on second-order neurons in the trigeminocervical complex (TCC). These neurons in turn project in the quintothalamic tract and,... |
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 | Figure 14-2 Positron emission tomography (PET) activation in migraine. In spontaneous attacks of episodic migraine there is activation of the region of the dorsolateral pons; an identical pattern is found in chronic migraine (not shown). This area, which includes the noradrenergic locus coeruleus, is fundamental to the expression of... |
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 | Figure 14-3 Posterior hypothalamic gray matter activation on positron emission tomography (PET) in a patient with acute cluster headache (A). (From A May et al: Lancet 352:275, 1998.) High-resolution T1 weighted MRI obtained using voxel-based morphometry... |
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 | Figure 14-4 MIDAS Questionnaire. |
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 | Figure 14-5 Magnetic resonance image showing diffuse meningeal enhancement after gadolinium administration in a patient with low CSF volume headache. |
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